ויאגרה כטיפול בסרטן

ישנם מספר צמחים מאוד טובים לערמונית מוגדלת וסרטן הערמונית: שורש סרפד http://www.ncbi.nlm.nih.gov/pubmed/10705733 דקל ננסי http://www.ncbi.nlm.nih.gov/pubmed/19067321 סוגי ערברבה http://www.rene-caisse.net/maria-treben-herbs/small-flowered-willow-herb-epilobium-parviflorum.html http://www.ncbi.nlm.nih.gov/pubmed/16492533 http://www.ncbi.nlm.nih.gov/pubmed/9140222 מיץ רימונים: http://pubs.acs.org/doi/abs/10.1021/jf071303g ללא ספק לצמחים אלו יש השפעה מהירה ויעילה לטיפול בבעיות ערמונית

מחקר מאונ' קולומביה מראה יכולת נגד סרטן הפרוסטטהוסרטן השד. החמור מופק מקליפות הדרים ,מה שאומר שכדאי להוסיף קליפות לימון מגורדות ללימונדה והסלט. קליפות הדרים גם מכילות לימונן ,חומר אנטי סרטני יעיל ולא רעיל

Proc Natl Acad Sci U S A. 2010 Sep 30. Sildenafil increases chemotherapeutic efficacy of doxorubicin in prostate cancer and ameliorates cardiac dysfunction. Das A, Durrant D, Mitchell C, Mayton E, Hoke NN, Salloum FN, Park MA, Qureshi I, Lee R, Dent P, Kukreja RC. Division of Cardiology, Department of Internal Medicine, Virginia Commonwealth University Pauley Heart Center and Department of Biochemistry and Molecular Biology, Virginia Commonwealth University Medical Center, Richmond, VA 23298. Abstract We have shown that the potent phosphodiesterase-5 (PDE-5) inhibitor sildenafil (Viagra) induces a powerful effect on reduction of infarct size following ischemia/reperfusion injury and improvement of left ventricular dysfunction in the failing heart after myocardial infarction or doxorubicin (DOX) treatment. In the present study, we further investigated the potential effects of sildenafil on improving antitumor efficacy of DOX in prostate cancer. Cotreatment with sildenafil enhanced DOX-induced apoptosis in PC-3 and DU145 prostate cancer cells, which was mediated by enhanced generation of reactive oxygen species, up-regulation of caspase-3 and caspase-9 activities, reduced expression of Bcl-xL, and phosphorylation of Bad. Overexpression of Bcl-xL or dominant negative caspase 9 attenuated the synergistic effect of sildenafil and DOX on prostate cancer cell killing. Furthermore, treatment with sildenafil and DOX in mice bearing prostate tumor xenografts resulted in significant inhibition of tumor growth. The reduced tumor size was associated with amplified apoptotic cell death and increased expression of activated caspase 3. Doppler echocardiography showed that sildenafil treatment ameliorated DOX-induced left ventricular dysfunction. In conclusion, these results provide provocative evidence that sildenafil is both a powerful sensitizer of DOX-induced killing of prostate cancer while providing concurrent cardioprotective benefit